Intuniv for ADHD: Help With Stimulant Side Effects
March 20, 2011ADHD Medication Problems: Why They Don’t Work
April 11, 2011Image via Wikipedia
ADHD Solutions Evolve With NeuroScience Evidence
About a year ago I traveled out to Sacramento, San Jose and San Francisco for three days of speaking to medical professionals who focus on treating ADHD, – and loved the opportunity to discuss the many new insights on the evolving pharma side of ADHD life – specifically Intuniv.
For example: Intuniv, as reported here at CorePsych Blog and elsewhere, is an interesting medication that announces a completely different approach to ADHD treatment with a non-stimulant application beyond AMP and MPH products. The news on Intuniv is most interesting, as it brings to the treatment table pharmacologic insights into brain function associated with executive function in the pre-frontal cortex mediated not by dopamine, not by norepinephrine, but by glutamate, a completely new neurotransmitter on ADHD treatment radar. There's simply more at stake than quickly meets the eye on a brief office visit.
NeuroScience Adds Additional ADHD Insights
ADHD is indeed more complicated than first glance at the current fascination with diagnostic appearances so far away from the underlying neurophysiology. Reporting here at CorePsych Blog we have been posting you on so many of these interesting new paths from SPECT brain imaging to a variety of other neurotransmitters including PEA that can significantly address ADHD symptoms – if, quite simply, they are measured precisely. – Which brings us to the NeuroScience perspective.
Of the labs in the country seeking deeper, more cellular and intracellular, answers to the complexity of brain function in general, and specifically with ADHD, NeuroScience stands out as a significant thought leader with applied neuroscience answers that translate on both a clinical and practical, economic level for those suffer with a variety of brain challenges. Recently I posted a brief report on 171 subsets of biomedical conditions that can significantly contribute to ADHD symptoms to punctuate the complexity of new scientific advances in brain evidence regarding ADHD.
Meeting In San Francisco
With all these new investigations and ADHD progress on both the pharma and the laboratory levels it's a special pleasure for me to have the opportunity to present some of these new findings with the NeuroScience team – Dr Gottfried Kellerman and Dr Sirid Kellerman – April 30 at an all day meeting at the Hotel Vitale on Mission Street, at the Embarcadero, in San Francisco. Dr Gottfried Kellerman, the founder and CEO of NeuroScience, and Dr Sirid Kellerman, his daughter and a molecular/cellular immunologist, are pioneers in the application of Neuro-Endo-Immune [NEI] SuperSystem assessments, from ADHD to brain injury and Autism. Two cases I will present cover the complexity of Autism and Asperger's, and every case presents an appearance of ADHD with the details of the underlying neurophysiology of NEI findings. Contact Christina Cowger [at the Events Calender on this CorePsych Blog page] if you are a professional on the West Coast thinking about adding to your professional quiver some diagnostic and treatment arrows that work more predictably for ADHD than “just try this.”
Hope to see you there! – And if you can't make it I'll keep you posted on the details here,
cp
Related articles
ADHD and PEA – The Stealth Neurotransmitter (corepsychblog.com)
When Your Spouse Has Adult ADHD (everydayhealth.com)
FDA Approves New ADHD Medication; Intuniv (medicalnewstoday.com)
6 Comments
David Summers, a 37 year old MS patient from Murfreesboro, Tennessee was a score of 8.0 on the Expanded Disability Status Scale (EDSS) when he had the Combination Liberation Therapy and Stem Cell Transplantation at CCSVI Clinic in March of 2012. Having been diagnosed in 1996 he had been in a wheelchair for the past decade without any sensation below the waist or use of his legs.“It was late 2011 and I didn’t have much future to look forward to” says David. “My MS was getting more progressive and ravaging my body. I was diagnosed as an 8.0 on the EDSS scale; 1 being mild symptoms, 10 being death. There were many new lesions on my optic nerves, in my brain and on my spinal cord. My neurologist just told me: ‘be prepared to deteriorate’. I knew that he was telling me I didn’t have much time left, or at least not much with any quality.” David had previously sought out the liberation therapy in 2010 and had it done in a clinic in Duluth Georgia. “The Interventional Radiologist who did it told me that 50% of all MS patients who have the jugular vein-clearing therapy eventually restenose. I didn’t believe that would happen to me if I could get it done. But I have had MS for 16 years and apparently my veins were pretty twisted up”. Within 90 days, David’s veins had narrowed again, and worse, they were now blocked in even more places than before his procedure.“I was so happy after my original procedure in 2010. I immediately lost all of the typical symptoms of MS. The cog fog disappeared, my speech came back, the vision in my right eye improved, I was able to regulate my body temperature again, and some of the sensation in my hands came back. But as much as I wanted to believe I felt something, there was nothing below the waist. I kind of knew that I wouldn’t get anything back in my legs. There was just way too much nerve damage now”. But any improvements felt by David lasted for just a few months.After his relapse, David and his family were frustrated but undaunted. They had seen what opening the jugular veins could do to improve him. Because the veins had closed so quickly after his liberation procedure, they considered another clinic that advocated stent implants to keep the veins open, but upon doing their due diligence, they decided it was just too risky. They kept on searching the many CCSVI information sites that were cropping up on the Internet for something that offered more hope. Finding a suitable treatment, especially where there was no known cure for the disease was also a race against time. David was still suffering new attacks and was definitely deteriorating. Then David’s mother Janice began reading some patient blogs about a Clinic that was offering both the liberation therapy and adult autologous stem cell injections in a series of procedures during a hospital stay. “These patients were reporting a ‘full recovery’ of their neurodegenerative deficits” says Janice, “I hadn’t seen anything like that anywhere else”. She contacted CCSVI Clinic in late 2011 and after a succession of calls with the researchers and surgeons they decided in favor of the combination therapies.For more information please visit http://www.ccsviclinic.ca/?p=904
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Tom-
You’re on the right track… off Prozac and NT testing will help a great deal as you will see other possible contributions to the ADHD presentation. Luvox is clean on 2D6, but blocks 3A4 thru which the Intuniv passes. The four you mentioned are the best choices and if they don’t work something else is amiss.
cp
Are these the best SSRI meds to be on in anticipation of being on Vyvanse and/or Intuniv: Effexor, Pristiq, Lexapro, and Celexa??? Any others? Which would have the least side-effect on already present hyperactivity and impulsivity? Currently my son is on 20mg of Prozac. We are going to do some neurotransmitter testing but want to move him off of Prozac asap in anticipation of moving him to Intuniv and/or Vyvanse.
I had a post related to this over on the testing options page…
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